Rouzier Clinical Intelligence
Rouzier Clinical IntelligencePhysician Intelligence

How it works

Clinical intelligence, not another chatbot

Organized medical knowledge so physicians can reach useful, cited answers in under 30 seconds — with progressive depth when you need it.

Intelligence network connecting research, evidence analysis, Rouzier commentary, and clinical application

Workflow

Three steps to a cited answer

  1. 01

    Search published evidence

    Studies with design, population, outcomes, and source links.

  2. 02

    Read the intelligence layer

    Strength, limitations, disagreement, and clinical relevance.

  3. 03

    Check Rouzier teaching

    Commentary, webinar chapters, and transcripts when available.

Three layers

Published evidence, scored and taught

Each layer adds context. Search starts with the literature, passes through structured evidence assessment, and connects to Dr. Rouzier's teaching when available.

  • 15s

    Bottom-line takeaway

    Abstract lead + rubric signal

  • 1 min

    Structured summary

    Design, bias, and relevance breakdown

  • 5 min

    Full study workspace

    Comparisons, citations, and teaching links

  • Layer 1

    Published evidence

    PubMed-linked studies with original sources.

  • Layer 2

    Evidence intelligence

    Bias, disagreement, and relevance scoring.

  • Layer 3

    Rouzier intelligence

    Webinars, commentary, and clinical teaching.

Clinical intelligence

Product demonstration

See the intelligence layer in action

Sample data
Popular
Fertility and Sterility2022

Testosterone therapy and cardiovascular risk: advances and controversies

Contemporary synthesis does not support categorical cardiovascular harm from appropriately indicated testosterone therapy.

Evidence: HighRelevance: High
Sample trial card2023

TRAVERSE: cardiovascular safety of testosterone in hypogonadal men

Large randomized cardiovascular-safety evidence for testosterone in a high-risk hypogonadal population.

Evidence: HighRelevance: High
JAMA (observational sample)2013

Association of testosterone therapy with mortality, MI, and stroke in men with low testosterone

An influential observational signal that raised CV concern and later required careful methodological critique.

Evidence: ModerateRelevance: High

Evidence answer

For appropriately selected hypogonadal men, current higher-quality evidence does not support a blanket increase in major cardiovascular events from testosterone replacement. Risk still depends on indication, formulation, monitoring, and baseline cardiovascular status.

HIGH

Evidence strength

Higher-quality contemporary trials outweigh older observational signals.

Study designHigh
Clinical relevanceModerate
Risk of biasLow

Evidence rubric · vv1

Evidence assessment rubric

Every indexed study receives a structured assessment derived from PubMed metadata and the abstract. Scores are heuristic signals for triage — not clinical recommendations.

Scores on every study

Evidence strength

Composite score reflecting study design, sample size, bias risk, and clinical relevance.

0.0 – 1.0 (higher = stronger design signals)

Clinical relevance

How directly the abstract and MeSH terms point to patient-centered outcomes in hormone medicine.

0.0 – 1.0

Risk of bias

Estimated confounding, selection, and design limitations inferred from publication type and abstract language.

0.0 – 1.0 (higher = more concern)

How evidence strength is calculated

Evidence strength = (design × 0.45) + (sample × 0.20) + ((1 − bias) × 0.20) + (relevance × 0.15)

  • Study design

    45%

    Classified from PubMed publication types — systematic reviews and RCTs score highest; case series and expert opinion score lowest.

  • Sample strength

    20%

    Estimated from sample-size patterns in the abstract (n=, participants, patients). Unclear samples receive a neutral score.

  • Risk of bias

    20%

    Inverted in the composite formula. Observational designs, retrospective language, and sponsorship signals increase bias risk.

  • Clinical relevance

    15%

    Keyword and MeSH signals for outcomes physicians care about — cardiovascular events, symptoms, fracture, mortality — vs. surrogate endpoints.

Study design tiers

Systematic review / meta-analysis0.92
Randomized controlled trial0.88
Prospective cohort0.72
Retrospective cohort0.58
Case-control study0.50
Narrative review0.55
Cross-sectional study0.42
Case series / case report0.30
Expert opinion / editorial0.25
Unclassified0.40

Progressive depth

  • 01

    15-second view

    Bottom-line takeaway from the abstract lead sentences.

  • 02

    1-minute view

    Abstract summary plus rubric dimension breakdown.

  • 03

    5-minute view

    Strengths, weaknesses, and limitations with explicit metadata caveats.

Important

  • Assessments are generated from PubMed metadata and abstracts only — full-text review may change interpretation.
  • Heuristic scoring is not a substitute for physician judgment or Dr. Rouzier's clinical teaching.
  • Draft assessments require editorial review before production publication when imported without auto-publish.
  • Rouzier commentary and webinar teaching are separate layers and may agree or disagree with study conclusions.

Ready to search with confidence?

From $129/month. Cancel anytime.