Research · PMID 42291041

PubMed sourcedAbstract-based analysis

Reviewed by Neal Rouzier, MD, Preventive Medicine · Sep 2026

Incremental prognostic value of coronary artery calcium progression within a large community-benefit calcium score registry.

American journal of preventive cardiology · June 2026 · Michael D Glidden, Santosh K Sirasapalli, Mark Yoder, Jean-Eudes Dazard, Zhuo Chen, et al.

What this study found

Serial CAC imaging may provide incremental prognostic value in individuals with pre-existing atherosclerosis. While incident CAC after a baseline zero score may confer limited short-term risk, it may present a window for preventive intervention.

retrospective_cohortNCT04075162

How RAGMD uses this study

Clinical questions this paper informs

Synthesis lives on the evidence question — this page shows where the study sits in that analysis.

Core study

Should coronary artery calcium scoring guide statin decisions?

Coronary artery calcium can reclassify intermediate-risk patients and inform statin initiation when risk is uncertain. A score of zero may support deferring therapy in selected low-risk individuals with shared decision-making.

Open evidence analysis

Supporting literature

Is ApoB more useful than LDL-C for cardiovascular risk prediction?

Prospective cohorts and meta-analyses suggest ApoB may capture atherogenic particle burden better than LDL-C alone, particularly when triglycerides are elevated or LDL particles are discordant with LDL-C.

Open evidence analysis

Supporting literature

Should adults take statins for primary cardiovascular prevention?

Meta-analyses of primary prevention trials show reduced major adverse cardiovascular events with statins in appropriately selected moderate-to-high-risk adults. Shared decision-making should weigh absolute risk reduction against side effects and patient preference.

Open evidence analysis

Supporting literature

Does elevated lipoprotein(a) independently increase cardiovascular risk?

Mendelian randomization and epidemiologic data support Lp(a) as a causal cardiovascular risk factor. Specific Lp(a)-lowering therapies are emerging but routine treatment beyond standard risk factor control awaits outcome trial confirmation.

Open evidence analysis

Supporting literature

Do therapies that raise HDL reduce cardiovascular events?

Trials of niacin and CETP inhibitors raised HDL without consistent reduction in cardiovascular events when added to statins, challenging the hypothesis that raising HDL alone is protective.

Open evidence analysis